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Live hemodynamics

12-lead ECG

LIVE
Ventricular rate
Sinus Rhythm
75bpm
Peripheral BP126/78mmHg
MAP92mmHg
LVEDP
10mmHg
Mean wedge
8mmHg
Forward output5.5L/min
Traces & panels 16 visible
ECG
Pressure tracings
Oxygenation
Additional panels
LIVEECG: mV · pressures: mmHg
IIECG lead II
75bpm · selected
V1ECG lead V1
mV±1.5 mV
ARTSystemic artery
126/78Peripheral Sys / Dia
Mean92mmHg
RHC pressures · mmHg
RARight atrium
4Mean · mmHg
RVRight ventricle
25/5Sys / EDP
Mean7mmHg
PAPulmonary artery
25/10Sys / Dia
Mean14mmHg
PAWPWedge
8Mean · mmHg
AoAortic reference
120/80Sys / Dia
Mean92mmHg
LVLeft ventricle
120/10Sys ref. / EDP
Mean43mmHg
FiO₂ 21%Resp ↑ in / ↓ out
Pulse oximetry
98SpO₂ % · simulated
Hemodynamic OverlayCompare traces
LEFT-HEART PRESSURES

LV → LVOT → Aorta

LVEDP10mmHg
LV cavity
Mean43mmHg
LV outflow tract
Mean43mmHg
Aorta
Mean92mmHg
IABP offLV reference 120/10 mmHgAo reference 120/80 mmHgReference peak-to-peak Δ 0 mmHg
Pullback interpretation & assumptions

This preset has no assumed LV outflow gradient. Aortic diastolic pressure rises relative to LV diastolic pressure as the catheter enters the aorta. These are synthetic left-heart pressures with assumed gradients and LVEDP, not measurements inferred from RHC. Sequential peak-to-peak values are not simultaneous instantaneous or Doppler gradients. Readouts show reference systolic/diastolic pressures, not augmented diastolic peaks. During Impella or ECMO support, the aortic readout instead shows time-averaged MAP; its dashed guide marks that mean. IABP adds the same timed aortic augmentation as ART; deflation reduces the following LV/LVOT systolic peak by an illustrative 8 mmHg. LVEDP follows the current disease, compensation, rate and intervention model independently of wedge pressure. Mitral stenosis or MR v waves can elevate wedge without an equivalent LVEDP rise. The dashed guide marks LVEDP immediately before contraction; minimum diastolic pressure is lower. Restriction/constriction add an early dip and plateau unless tamponade is selected. Assistance follows the selected 1:1, 1:2 or 1:3 ratio. Balloon inflation does not create an LV diastolic pressure hump. Switching Off restores the unsupported model. The optional PVC capture illustrates post-extrasystolic LV/aortic changes separately from this sequential recording. Changing controls clears the recording.

1. Atrial systole2. Isovolumic contraction3. Rapid ejection4. Reduced ejection5. Isovolumic relaxation6. Rapid filling7. Reduced filling / diastasis
Cardiac cycle
Live cursor
Wiggers diagram
Ao · mean 92LV · mean 45LA · mean 9mmHgVolume / sounds

LV pressure–volume

Live comparison · illustrative volumes
Normal referenceUntreated
1 · MC2 · AO3 · AC4 · MO
Forward stroke volume— mL/beat
JVP
CHEST RADIOGRAPH SYNTHETIC · PA
RL
No modeled edemaPAWP 8 mmHgNo assumed cardiomegaly
IVC17 mm · 62% sniff collapse
17 mmILLUSTRATIVE
Normal size · ≥50% collapse
CXR model

Illustrative opacities begin above a transmural filling-pressure proxy of 20 mmHg and increase to 40 mmHg. With PEEP, recruitment improves aeration and lower transmural filling reduces modeled congestion; transmitted airway pressure is excluded from the opacity estimate. PAWP below the image remains the measured pressure. This depicts a simplified later equilibrium, not immediate lung-fluid removal. Real edema depends on acuity, permeability and lymphatic adaptation; it may lag pressure changes. Cardiac silhouette reflects assumed chronic remodeling; pulmonary hypertension adds central vessel prominence and peripheral pruning. Effusions are illustrative at high congestion. No ARDS or immediate radiographic response is inferred clinically. This is not a predicted patient X-ray. Radiographic patterns · Positive pressure.

Acute MR and unilateral edema ↗

ESTIMATED FICK OUTPUT5.5L/min

Simulated forward flow 5.5 L/min · Fick difference 0%

Oxygen values & calculation
Fick method reference ↗ · Assumed VO₂ limitations ↗
CARDIAC OUTPUT

Thermodilution 6-second measurement · 5× replay

0s10s20s30s0−0.5°C
—L/minAwaiting complete curve
Sinus rhythm · 75 bpm← 6 seconds →

Hemodynamic summary

mmHg · waveform measurements
ReferenceMore abnormal
LocationSysDiaMean
Right atrium814
Right ventricle2407
Pulmonary artery24814
Wedge / LA proxy1258
Left ventricle123543
Central aorta1218092
LVEDP10mmHg · reference
Mean wedge8mmHg · waveform mean
RVEDP5mmHg · reference
Cardiac output5.5L/min · modeled forward flow
Fick · estimated5.5 L/min
Thermodilution— L/min
Resistancedyn·s/cm⁵WU
SVR127616.0
PVR821.0
Definitions, calculations & colour

Numbers shift smoothly from black toward crimson and become bolder as they move farther outside their reference range or show greater waveform abnormality. The colour spectrum is an illustrative teaching cue, not a validated severity grade, diagnosis or treatment trigger.

Illustrative alert landmarks: mean PA ≥20; mean PCWP ≥20; LVEDP ≥20 mmHg; PVR >2 WU (>160 dyn·s/cm⁵). These landmarks do not cause abrupt colour changes or mean “very severe” disease: the ESC PH definition is mean PA >20 mmHg, and PCWP 16–19 is already above the ESC reference and gains progressively more emphasis.

Other alert landmarks: RA mean/minimum ≥15 or peak ≥20; RV peak ≥60 or RVEDP ≥15; PA peak ≥60 or minimum ≥25; wedge peak ≥30 or minimum ≥20; LV peak <80 or ≥180, minimum ≥20; Ao peak <90 or ≥180, minimum <40 or ≥110, mean <65 or ≥130 mmHg. CO <3 or >8 L/min; SVR <600 or ≥1600 dyn·s/cm⁵ (<7.5 or ≥20 WU). Apart from the ACC low-pressure warning criteria, these cutoffs are illustrative. IABP-augmented peaks can trigger alerts.

Atrial peaks/troughs follow their mean pressure, with extra emphasis for extreme waves. Ventricular minimum/mean colours reflect systolic and EDP abnormalities; no standalone normal ventricular mean is implied. Systemic mean colour also reflects abnormal systolic/diastolic pressure.

ESC/ERS 2022, Table 11 supplies available normal RHC ranges. ACC shock guidance (2025) identifies SBP <90 or MAP <65 as warning criteria and PCWP/LVEDP >15 as LV congestion in shock. Shock also requires evidence of hypoperfusion; it is not diagnosed by the table colour. Supplemental ventricular, arterial and SVR ranges are identified in the reference panel.

Pressures are displayed to the nearest whole mmHg; flow and Wood-unit resistance use one decimal place. Calculations, waveform generation and colour emphasis use the unrounded values. Small differences can result if you calculate from the rounded display.

Forward modeled CO. SVR = (mean Ao − mean RA) ÷ CO; PVR = (mean PA − mean wedge) ÷ pulmonary flow. Pulmonary flow equals systemic flow only without shunts or bypass. Qp includes net shunt flow and excludes the ECMO bypass. 1 Wood unit = 80 dyn·s/cm⁵. Resistance is undefined at zero output.

Ao is central aortic pressure. The peripheral ART trace has modest pulse-pressure amplification and transit delay, with approximately preserved mean pressure. This is an illustrative radial waveform transfer, not a patient-specific conversion. Arterial waveform reference.

Updates with conditions, rhythm, rate and IABP. Sys/Dia show the maximum/minimum over the modeled rhythm sequence; Mean is the time average. IABP augmentation is included. For NSVT, peak and trough can come from different beats; the full repeating sequence includes baseline rhythm and VT bursts. Flow is averaged over that same sequence.

For RA and wedge, Sys/Dia represent waveform peaks/troughs, not ventricular systole/diastole. Ventricular Dia is the minimum, not EDP. Wedge estimates LA pressure, not LVEDP.

Mean wedge is an LA pressure proxy, not LVEDP. A large v wave may raise the mean; wedge–LV timing must be aligned before judging a mitral gradient. Wedge and LVEDP explained ↗

Circulation Studio · Tutorial

Take the intermediate simulator tour.

This tour assumes basic cardiovascular vocabulary and starts with severe aortic stenosis before moving through paused waveforms and interventions. New to the terms? Begin with Foundations.

Beginner foundationsAt your pace